37th Hemophilia Symposium by I. Scharrer (auth.), Professor Dr. med. Inge Scharrer,

By I. Scharrer (auth.), Professor Dr. med. Inge Scharrer, Professor Dr. med. Wolfgang Schramm (eds.)

This e-book comprises the contribution to the thirty seventh Hemophilia Symposium, Hamburg 2006. the most issues are epidemiolgy, remedy of inhibitors in hemophiliacs, hemophilic arthropathy and synovitis, correct hemophilia remedy 2006, and pediatric hemostasiology. the quantity is rounded off by way of quite a few unfastened papers and posters on hemophilia, casuistics, and diagnostics.

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The large group of patients on demand certainly includes many patients using only sporadically factor substitutes. The statistics of the annual amount of substitutes reveals that only 418 (54%) of all 778 patients regularly draw products. Intermediate prophylaxis is defined as prophylactic substitution over a short period of time. The status of 7% patients remains unknown. Hemophilia Registry of the Medical Commitee of the Swiss Hemophilia Association Fig. 3. Mode of substitution. ©AEK-Online-Register, Nov.

Each spot represents a single newly differentiated anti-FVIII antibody-producing plasma cell. The percentage of antigen-specific cells related to total IgG-producing cells as well as the limit of detection was calculated for each patient Fig. 7. Detection of FVIII-specific memory B cells in the peripheral blood of 6 patients with severe hemophilia A without FVIII inhibitors. HSA was used as negative control. The percentage of antigen-specific cells related to total IgG-producing cells as well as the limit of detection was calculated for each patient Detection of Factor VIII-Specific Memory B Cells in Patients with Hemophilia A and ...

11 12 W. Schramm and H. Krebs Mortality from Liver Disease and Cancer Therewith in the 2005/2006 survey the increase of liver disease as a cause of death has reached statistical significance the fourth time consecutively. 8%). 025), suggesting an ongoing brace of this trend in future (see Fig. 6d). The obvious reason for this probably can be attributed to the increasing number of deaths induced by liver cirrhosis and hepatocellular carcinoma due to chronic HCV [4]. As we still did not discriminate type of cancer in our surveys up to now there might be a relevant portion of patients in this group having died of primary hepatocellular carcinoma induced by chronic HCV intensifying the impact of liver disease on causes of death in patients with hemophilia even more.

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